Resting-State Functional Connectivity Changes in Prodromal Individuals Progressing to Parkinson’s Disease

Researcher(s)

  • Kaitlyn McKenna, Neuroscience, University of Delaware

Faculty Mentor(s)

  • Roxana Burcui, KAAP, University of Delaware

Abstract

 

Kaitlyn McKenna 1 and Roxana G. Burciu, Ph.D. 2

 

1 Department of Psychological & Brain Sciences, University of Delaware, Newark, DE

2 Department of Kinesiology and Applied Physiology, University of Delaware, Newark, DE

 

Parkinson’s disease (PD) is a progressive neurodegenerative disorder that begins years

before motor symptoms emerge. By the time of diagnosis, over half of the brain’s

dopaminergic neurons are already lost. The prodromal phase is when individuals exhibit

subtle symptoms or risk factors without meeting diagnostic criteria – it offers a critical

window for early detection and intervention. However, the brain changes that

differentiate those who eventually convert to PD from those who remain at risk are

poorly understood. In this pilot study, we leveraged longitudinal data from the

Parkinson’s Progression Markers Initiative (PPMI), a large international study supported

by the Michael J. Fox Foundation, to investigate brain changes in individuals at risk for.

We analyzed resting-state fMRI data from two timepoints (baseline and 2-year

follow-up) in 13 prodromal participants. Six converted to PD shortly after year 2, while

seven did not. The two groups had similar sociodemographic characteristics at baseline.

We conducted seed-based voxelwise analyses of resting-state fMRI data using the left

and right putamen – key regions presenting functional changes in PD. At baseline, there

were no detectable differences in the local connections within the basal ganglia or

cortical motor regions. By year 2, converters exhibited reduced local connectivity within

the basal ganglia combined with increased connectivity between the putamen and

cortical regions, including motor regions. These changes, not seen in non-converters,

may reflect early network reorganization, preceding symptom onset. Converters showed

more prodromal symptoms and risk factors, including family history, with no mutations in

known PD genes in either group. These resting-state connectivity changes and risk

profiles may serve as early biomarkers of conversion but require validation in larger

cohorts. Our findings highlight the potential of multimodal approaches – combining

imaging, clinical, and genetic data – to detect Parkinson’s disease in its earliest stages.